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  #21  
Old 03-30-2012, 01:47 PM
crimson n' camo crimson n' camo is offline
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Originally Posted by B100888 View Post
You betcha!! If they did not come from tested farms. Thats why everything is being turned over to the best departments to make sure all is done and done right. Just another stupid question!!! Next?

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  #22  
Old 03-30-2012, 04:46 PM
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Originally Posted by sandbur View Post
terry-In the middle of this post, it says the "webmater" feels CWD originated at the Foothills Research Station. Who was this wemaster? What are or were his credentials?

I believe that Terry is the "webmaster" in question and that his warning of webmaster opinion is a signal for leaving the realm of documented science and traveling into the world of creative fiction.
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  #23  
Old 03-30-2012, 06:05 PM
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Default Munsterlndr wrong again

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Originally Posted by Munsterlndr View Post
I believe that Terry is the "webmaster" in question and that his warning of webmaster opinion is a signal for leaving the realm of documented science and traveling into the world of creative fiction.



wrong again Munsterlndr. that is NOT my site, and i am NOT the webmaster of mad-cow.org/. Dr. Tom Pringle was. the site has been dead for years. no fantacy, this is FACT. as i stated in my (dare i say blog, because the science there is so damning for deer farmers). but like i said there ;



UPDATE ON SOME OLD HISTORY ON CWD AND GAME FARMS AND ANIMAL ESCAPES, BEING TRADED FROM STATE TO STATE ETC. ...


http://chronic-wasting-disease.blogs...n-dnr-not.html



you can see Dr. Thomas Pringle's work here ;


http://www.ncbi.nlm.nih.gov/pubmed?t...22%5BAuthor%5D





Quote:
Originally Posted by Munsterlndr View Post
If you are implying that transferring animals among game farms in 1998 was the source of the Wisconsin outbreak, as you appear to be speculating above, it's highly, highly unlikely that was the case. The geographic and demographic patterns of the Wisconsin outbreak indicate that it was most likely introduced into Wisconsin, some time in the 1960's and by far the most likely cause was deer brought back from out west by hunters that were improperly disposed of. An introduction as recent as 1998 just does not fit the modeling programs that have been developed, based on the spread of the disease. If that was the case the outbreak would be much more localized and have a much lower prevalence rate.



highly unlikely is your opinion. it is not fact.


Dr. Tom Pringle site mad-cow.org/ has a lot of great history on the facts of Transmissible Spongiform Encephalopathy TSE prion disease, when it was up and running.


now who is conjecturing what? your just flat our wrong there Munsterlndr, like it or not. i can't believe you have gone and started just making stuff up now


Dr. Thomas Pringle is well known around the world for his work on mad cow disease, cwd, scrapie, and all TSE prion disease. he's got some good stuff on Alzhiemer's disease, and other disease. holy mad cow, i can't believe you did not know this, or are you just conjecturing again there Munsterlndr, or are you just making stuff up now like some of the deer farmers now?





A CONVERSATION WITH: Thomas Pringle; A 24-Hour Lab Meeting on Mad Cow Illness

By SANDRA BLAKESLEE Published: March 13, 2001

Correction Appended



Sign In to E-Mail Print



Dr. Thomas Pringle, a biochemist, never planned to become an authority on mad cow disease.

After years of teaching college biology, an inheritance allowed him to set up a small foundation dedicated to environmental issues in the Pacific Northwest. He became interested in chronic wasting disease -- a related malady in deer and elk -- and that led him to mad cow disease.

Today Dr. Pringle, 55, runs the Sperling Biomedical Foundation out of his home in Eugene, Ore., gathering information on transmissible spongiform encephalopathies, including mad cow disease, chronic wasting disease, the human ailment Creutzfeldt-Jakob disease or C.J.D., and a new variant of C.J.D., which people contract from eating affected beef. All are believed to be caused by aberrant proteins called prions.

''My main interest is in annotating the prion gene family within the Human Genome Project,'' Dr. Pringle said recently. The project recently identified more than 30,000 human genes and published their DNA sequences on public databases. Each day, Dr. Pringle's prion disease Web site (www.mad-cow.org) collects articles and research papers written about these diseases, from this country and abroad. ''It is intended as a model for intensive annotation of any supergene family,'' he said. ''I believe it is ethically important for scientists to inform public debate on highly complex topics with major policy components.''


snip...(don't have a mad cow anti-snippers, not enough room to post complete article)



but for the conjecturer , here is the full article and interview with Dr. Thomas Pringle about mad-cow.org/ with NYT ;



http://www.nytimes.com/2001/03/13/sc...ted=all&src=pm




Dr. Thomas Pringle



https://www.google.com/webhp?hl=en&t...w=1067&bih=537




kind regards, terry
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  #24  
Old 03-30-2012, 06:29 PM
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Default sandbur questions

Quote:
Originally Posted by sandbur View Post
terry-In the middle of this post, it says the "webmater" feels CWD originated at the Foothills Research Station. Who was this wemaster? What are or were his credentials?

Another question concerning the Wis outbreak. Is there any evidence of CWD infected tissues being incorporated into deer feed or other livestock feed that might have been fed to deer?



say there sandbur,


the author is Dr. Thomas Pringle. see my reply to Munsterlndr, about his conjecturering


i think, but i am not sure, if my mind don't fail me, could be another link, but there may be some information about CWD and the Foothills Research Station here, with the other stuff. i could be wrong now, but i remember a some information at the BSE Inquiry.





*** Spraker suggested an interesting explanation for the occurrence of CWD. The deer pens at the Foot Hills Campus were built some 30-40 years ago by a Dr. Bob Davis. At or abut that time, allegedly, some scrapie work was conducted at this site. When deer were introduced to the pens they occupied ground that had previously been occupied by sheep.


(PLEASE NOTE SOME OF THESE OLD UK GOVERNMENT FILE URLS ARE SLOW TO OPEN, AND SOMETIMES YOU MAY HAVE TO CLICK ON MULTIPLE TIMES, PLEASE BE PATIENT, ANY PROBLEMS PLEASE WRITE ME PRIVATELY, AND I WILL TRY AND FIX OR SEND YOU OLD PDF FILE...TSS)


http://collections.europarchive.org/...m11b/tab01.pdf


since then though, i thought i might throw this in ;


2011

Wednesday, October 12, 2011

White-tailed deer are susceptible to the agent of sheep scrapie by intracerebral inoculation



http://www.veterinaryresearch.org/co...716-42-107.pdf




http://chronic-wasting-disease.blogs...ptible-to.html




well sandbur, i been screaming about the CWD and feed from animal protein for a decade now. there is plenty of evidence, but horizontal transmission (in my opinion), with CWD, is more concerning. but about the mad deer and elk feed, i know game farmers used it to fatten up their deer fast, some hunters using bait piles probably did too. but here is some data on the mad deer and elk feed. oral feeding is a very sufficient mode of transmission CWD.

dagonit, i am going to have to get long winded again. the anti-bloggers don't want my blogs, where the following information is stored, so i must get long winded to answer sandburs question. if i snip, the anti-snippers will start crying, so, your just gonna have to digest these prions the long way continued
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  #25  
Old 03-30-2012, 06:51 PM
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Default sandbur questions part 2 continued

Quote:
Originally Posted by sandbur View Post
terry-In the middle of this post, it says the "webmater" feels CWD originated at the Foothills Research Station. Who was this wemaster? What are or were his credentials?

Another question concerning the Wis outbreak. Is there any evidence of CWD infected tissues being incorporated into deer feed or other livestock feed that might have been fed to deer?



well, that didn't work, because all the mad deer and elk feed that was banned that went out into commerce, some in Wisconsin, well, there was so much, i had 40,662 charactors, and it would take 4 or more continued parts. and the anti-long-winded-factors would complain. so where do i start ??? and how do i do this. ??? now, none of these fda warning letters urls work anymore. so ???


i am gonna have to snip. if you anti-snippers don't like it, i'm sorry.





Subject: ''MORE'' 'VIOLATORS' of Animal Proteins Prohibited in Ruminant Feed--U.S.A. (more and more MAD COW FEED RULES BROKEN IN U.S.A.]

Date: Tue, 24 Apr 2001 09:45:15 –0700

From: "Terry S. Singeltary Sr."

Reply-To: Bovine Spongiform Encephalopathy

To: BSE-L@uni-karlsruhe.de References: 1 , 2

######### Bovine Spongiform Encephalopathy #########

Greetings again List Members,

snip...

In addition, we note that you are using approximately 140 pounds of cracked corn to flush your mixer used in the manufacture of animal feeds containing prohibited material. This flushed material is fed to wild game including deer, a ruminant animal. Feed material which may potentially contain prohibited material should not be fed to ruminant animals which may become part of the food chain.

The above is not intended to be an all-inclusive list of deviations from the regulations. As a manufacturer of materials intended for animal feed use, you are responsible for assuring that your overall operation and the products you manufacture and distribute are in compliance with the law. We have enclosed a copy of FDA's Small Entity Compliance Guide to assist you with complying with the regulation... blah, blah, blah...

http://www.fda.gov/foi/warning_letters/g1115d.pdf


-------- Original Message --------

Subject: MAD DEER FEED BAN WARNING LETTER RECALL 6 TONS DISTRIBUTED USA

Date: Wed, 20 Oct 2004 14:53:56 –0500

From: "Terry S. Singeltary Sr." flounder@WT.NET

Reply-To: Bovine Spongiform Encephalopathy BSE-L@UNI-KARLSRUHE.DE

To: BSE-L@UNI-KARLSRUHE.DE

##################### Bovine Spongiform Encephalopathy #####################

PRODUCT

Product is __custom made deer feed__ packaged in 100 lb. poly bags. The product has no labeling. Recall # V-003-5.

CODE

The product has no lot code. All custom made feed purchased between June 24, 2004 and September 8, 2004.

RECALLING FIRM/MANUFACTURER

Farmers Elevator Co, Houston, OH, by telephone and letter dated September 27, 2004. Firm initiated recall is ongoing.

REASON

Feed may contain protein derived from mammalian tissues which is prohibited in ruminant feed.

VOLUME OF PRODUCT IN COMMERCE

Approximately 6 tons.

DISTRIBUTION OH.

END OF ENFORCEMENT REPORT FOR October 20, 2004

http://www.fda.gov/TSS

################# BSE-L-subscribe-request@uni-karlsruhe.de #################

now, just what is in that deer feed? _ANIMAL PROTEIN_

Subject: MAD DEER/ELK DISEASE AND POTENTIAL SOURCES

Date: Sat, 25 May 2002 18:41:46 –0700

From: "Terry S. Singeltary Sr."

Reply-To: BSE-L

To: BSE-L

8420-20.5% Antler Developer For Deer and Game in the wild Guaranteed Analysis Ingredients / Products Feeding Directions

snip...

_animal protein_

http://www.surefed.com/deer.htm

BODE'S GAME FEED SUPPLEMENT #400 A RATION FOR DEER NET WEIGHT 50 POUNDS 22.6 KG.

snip...

_animal protein_


http://www.bodefeed.com/prod7.htm

Ingredients

Grain Products, Plant Protein Products, Processed Grain By-Products, Forage Products, Roughage Products 15%, Molasses Products,

__Animal Protein Products__,



snip...


http://www.bodefeed.com/prod6.htm

===================================

MORE ANIMAL PROTEIN PRODUCTS FOR DEER

Bode's #1 Game Pellets A RATION FOR DEER F3153

GUARANTEED ANALYSIS Crude Protein (Min) 16% Crude Fat (Min) 2.0% Crude Fiber (Max) 19% Calcium (Ca) (Min) 1.25% Calcium (Ca) (Max) 1.75% Phosphorus (P) (Min) 1.0% Salt (Min) .30% Salt (Max) .70%

Ingredients

Grain Products, Plant Protein Products, Processed Grain By-Products, Forage Products, Roughage Products, 15% Molasses Products,

__Animal Protein Products__,


FEEDING DIRECTIONS Feed as Creep Feed with Normal Diet

http://www.bodefeed.com/prod8.htm

INGREDIENTS

Grain Products, Roughage Products (not more than 35%), Processed Grain By-Products, Plant Protein Products, Forage Products,


__Animal Protein Products__,



snip...



DIRECTIONS FOR USE

Deer Builder Pellets is designed to be fed to deer under range conditions or deer that require higher levels of protein. Feed to deer during gestation, fawning, lactation, antler growth and pre-rut, all phases which require a higher level of nutrition. Provide adequate amounts of good quality roughage and fresh water at all times.

http://www.profilenutrition.com/Prod...r_pellets.html

Subject: MAD COW FEED RECALL MI MAMMALIAN PROTEIN VOLUME OF PRODUCT IN COMMERCE 27,694,240 lbs

Date: August 6, 2006 at 6:14 pm PST


snip...


Subject: MAD COW FEED RECALL AL AND FL VOLUME OF PRODUCT IN COMMERCE 125 TONS Products manufactured from 02/01/2005 until 06/06/2006

Date: August 6, 2006 at 6:16 pm PST

PRODUCT a) CO-OP 32% Sinking Catfish, Recall # V-100-6; Performance Sheep Pell W/Decox/A/N, medicated, net wt. 50 lbs, Recall # V-101-6; c) Pro 40% Swine Conc Meal -- 50 lb, Recall # V-102-6; d) CO-OP 32% Sinking Catfish Food Medicated, Recall # V-103-6; e)

===============================================

"Big Jim's" BBB Deer Ration, Big Buck Blend, Recall # V-104-6; f)

===============================================



snipped ; out are the many other species that got this feed...tss



REASON Animal and fish feeds which were possibly contaminated with ruminant based protein not labeled as "Do not feed to ruminants".

VOLUME OF PRODUCT IN COMMERCE 125 tons DISTRIBUTION AL and FL

END OF ENFORCEMENT REPORT FOR AUGUST 2, 2006

###

http://www.fda.gov/bbs/topics/enforc.../ENF00963.html

CONTAINS NON-BINDING RECOMMENDATIONS Guidance for Industry SMALL ENTITIES COMPLIANCE GUIDE FOR FEEDERS OF RUMINANT ANIMALS WITHOUT ON-FARM FEED MIXING OPERATIONS1

FDA’s guidance documents, including this guidance, do not establish legally enforceable responsibilities. Instead, guidances describe the Agency’s current thinking on a topic and should be viewed only as recommendations, unless specific regulatory or statutory requirements are cited. The use of the word “should” in Agency guidances means that something is suggested or recommended, but not required.

http://www.fda.gov/downloads/AnimalV.../UCM052385.pdf

RECALLS AND FIELD CORRECTIONS: VETMED -- CLASS IIPRODUCT & CODES: Animal feed products, packaged in 5, 25, 50, and 55 pound bags, and in bulk, intended for both ruminant and non-ruminant animals. The products are as follows: Recall # V-195-1 through V-350-1.

RUMINANT FEED PRODUCTS:

RECALL NO. PRODUCT NO. PRODUCT NAME


snip...


17% Doe Pellet V-219-1 44350 B. 21% Buck Pellet V-220-1 44450

Legends Ranch Pellet V-221-1 44500

Legends 17% Breeder Pellet V-222-1 1652 B. Vitamin E-20 V-223-1 1614 B. Vitamin A-30 V-224-1 44550

Legends 17% Breeder Pellet V-225-1 44650 Legends 13.5% Rut Pellet V-226-1 44750 Deer Starter


snip...


Deer Premix (J) HFB V-246-1 49750 39% Lamb Premix (J) HFB V-247-1 49850 Lamb Starter Premix (J) HFB V-248-1 120850 Brood Cow Deluxe Mineral V-249-1 152850 B. A-D-E Mix

NON-RUMINANT FEED PRODUCTS:

snipped out the other species that got all this mad cow feed, not enough room. ...tss

MANUFACTURER: Buckeye Feed Mills, Dalton, Ohio. RECALLED BY: Manufacturer visited local customers on April 17, 2001. On April 18 and 19, 2001, manufacturer mailed and faxed recall notices. Firm initiated recall is ongoing.

DISTRIBUTION: Al, CT, DE, FL, GA, IL, IN, IA, KY, ME, MD, MA, MO, MN, MS, NH, NJ, NY, NC, OH, OR, PA, RI, TN, VA, WV, and WI.

QUANTITY: 2,790 tons of ruminant feed products and 14,000 tons of non-ruminant feed products. REASON: The animal feed products may contain protein derived from mammalian tissues.

http://www.fda.gov/bbs/topics/ENFORC.../ENF00694.html

http://www.legendsranch.com/feedsProgram.php?id=6



====================================

PRODUCT Custom deer feed made for a Wisconsin farm.

====================================


The product was in bags holding about 40 pounds each. Recall # V-122-4. CODE 1-30-04 on the product invoice and mixing record.

RECALLING FIRM/MANUFACTURER Crivitz Feed Mill, Crivitz, WI, by telephone on February 20, 2004. Wisconsin State initiated recall is complete.

REASON The recalled deer feed contained steamed bone meal which is prohibited material in feed for ruminants.

VOLUME OF PRODUCT IN COMMERCE 515 pounds.

DISTRIBUTION WI.

END OF ENFORCEMENT REPORT FOR APRIL 7, 2004

###

http://www.fda.gov/bbs/topics/enforc.../ENF00842.html

Ruminant animals are any animals with a four-chambered stomach including cattle, sheep, goats, buffalo, elk, and deer.


http://madcowfeed.blogspot.com/2010/...w-feed-in.html


http://transmissiblespongiformenceph...forcement.html


continued...
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  #26  
Old 03-30-2012, 06:58 PM
Terry S. Singeltary Sr.'s Avatar
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Default sandbur questions part 3...continued

Quote:
Originally Posted by Terry S. Singeltary Sr. View Post
well, that didn't work, because all the mad deer and elk feed that was banned that went out into commerce, some in Wisconsin, well, there was so much, i had 40,662 charactors, and it would take 4 or more continued parts. and the anti-long-winded-factors would complain. so where do i start ??? and how do i do this. ??? now, none of these fda warning letters urls work anymore. so ???


i am gonna have to snip. if you anti-snippers don't like it, i'm sorry.


continued...





-------- Original Message --------

Subject: DOCKET-- 03D-0186 -- FDA Issues Draft Guidance on Use of Material From Deer and Elk in Animal Feed; Availability

Date: Fri, 16 May 2003 11:47:37 –0500

From: "Terry S. Singeltary Sr." To: fdadockets@oc.fda.gov

Greetings FDA,

i would kindly like to comment on;

Docket 03D-0186

FDA Issues Draft Guidance on Use of Material From Deer and Elk in Animal Feed; Availability

Several factors on this apparent voluntary proposal disturbs me greatly, please allow me to point them out;

1. MY first point is the failure of the partial ruminant-to-ruminant feed ban of 8/4/97. this partial and voluntary feed ban of some ruminant materials being fed back to cattle is terribly flawed. without the _total_ and _mandatory_ ban of all ruminant materials being fed back to ruminants including cattle, sheep, goat, deer, elk and mink, chickens, fish (all farmed animals for human/animal consumption), this half *** measure will fail terribly, as in the past decades...

2. WHAT about sub-clinical TSE in deer and elk? with the recent findings of deer fawns being infected with CWD, how many could possibly be sub-clinically infected. until we have a rapid TSE test to assure us that all deer/elk are free of disease (clinical and sub-clinical), we must ban not only documented CWD infected deer/elk, but healthy ones as well. it this is not done, they system will fail...

3. WE must ban not only CNS (SRMs specified risk materials), but ALL tissues. recent new and old findings support infectivity in the rump or *** muscle. wether it be low or high, accumulation will play a crucial role in TSEs.

4. THERE are and have been for some time many TSEs in the USA. TME in mink, Scrapie in Sheep and Goats, and unidentified TSE in USA cattle. all this has been proven, but the TSE in USA cattle has been totally ignored for decades. i will document this data below in my references.

5. UNTIL we ban all ruminant by-products from being fed back to ALL ruminants, until we rapid TSE test (not only deer/elk) but cattle in sufficient numbers to find (1 million rapid TSE test in USA cattle annually for 5 years), any partial measures such as the ones proposed while ignoring sub-clinical TSEs and not rapid TSE testing cattle, not closing down feed mills that continue to violate the FDA's BSE feed regulation (21 CFR 589.2000) and not making freely available those violations, will only continue to spread these TSE mad cow agents in the USA. I am curious what we will call a phenotype in a species that is mixed with who knows how many strains of scrapie, who knows what strain or how many strains of TSE in USA cattle, and the CWD in deer and elk (no telling how many strains there), but all of this has been rendered for animal feeds in the USA for decades. it will get interesting once someone starts looking in all species, including humans here in the USA, but this has yet to happen...

6. IT is paramount that CJD be made reportable in every state (especially ''sporadic'' cjd), and that a CJD Questionnaire must be issued to every family of a victim of TSE. only checking death certificates will not be sufficient. this has been proven as well (see below HISTORY OF CJD -- CJD QUESTIONNAIRE)

7. WE must learn from our past mistakes, not continue to make the same mistakes...

REFERENCES

Oral transmission and early lymphoid tropism of chronic wasting disease PrPres in mule deer fawns (Odocoileus hemionus ) Christina J. Sigurdson1, Elizabeth S. Williams2, Michael W. Miller3, Terry R. Spraker1,4, Katherine I. O'Rourke5 and Edward A. Hoover1

Department of Pathology, College of Veterinary Medicine and Biomedical Sciences, Colorado State University, Fort Collins, CO 80523- 1671, USA1 Department of Veterinary Sciences, University of Wyoming, 1174 Snowy Range Road, University of Wyoming, Laramie, WY 82070, USA 2 Colorado Division of Wildlife, Wildlife Research Center, 317 West Prospect Road, Fort Collins, CO 80526-2097, USA3 Colorado State University Veterinary Diagnostic Laboratory, 300 West Drake Road, Fort Collins, CO 80523-1671, USA4 Animal Disease Research Unit, Agricultural Research Service, US Department of Agriculture, 337 Bustad Hall, Washington State University, Pullman, WA 99164-7030, USA5

Author for correspondence: Edward Hoover.Fax +1 970 491 0523. e-mail ehoover@lamar.colostate.edu



snip...


These results indicate that CWD PrP res can be detected in lymphoid tissues draining the alimentary tract within a few weeks after oral exposure to infectious prions and may reflect the initial pathway of CWD infection in deer. The rapid infection of deer fawns following exposure by the most plausible natural route is consistent with the efficient horizontal transmission of CWD in nature and enables accelerated studies of transmission and pathogenesis in the native species.

snip...

These findings support oral exposure as a natural route of CWD infection in deer and support oral inoculation as a reasonable exposure route for experimental studies of CWD.

snip...

http://vir.sgmjournals.org/cgi/content/full/80/10/2757


REFERENCES

snip...see full text ;


http://madcowfeed.blogspot.com/2008/...ues-draft.html



The finding of PrPCWD in several extraneural tissues including cardiac muscle and the endocrine system suggests that further investigation and monitoring of the potential transmissibility to other species including humans is warranted.

SNIP...


http://digitalcommons.unl.edu/cgi/vi...t=zoonoticspub


Wednesday, September 08, 2010

CWD PRION CONGRESS SEPTEMBER 8-11 2010

http://chronic-wasting-disease.blogs...rion-2010.html



http://www.ars.usda.gov/research/pub..._no_115=228787



Simian vCJD can be easily triggered in cynomolgus monkeys on the oral route using less than 5 g BSE brain homogenate.


http://www.prion2007.com/pdf/Prion%2...0Abstracts.pdf


see much more tonnage of recalled prohibited banned suspect BSE contaminated feed here ;


Saturday, November 6, 2010

TAFS1 Position Paper on Position Paper on Relaxation of the Feed Ban in the EU

Berne, 2010 TAFS INTERNATIONAL FORUM FOR TRANSMISSIBLE ANIMAL DISEASES AND FOOD SAFETY a non-profit Swiss Foundation

http://madcowfeed.blogspot.com/2010/...ion-paper.html

Archive Number 20101206.4364 Published Date 06-DEC-2010 Subject PRO/AH/EDR>

Prion disease update 2010 (11) PRION DISEASE UPDATE 2010 (11)

http://www.promedmail.org/direct.php?id=20101206.4364

Sunday, February 5, 2012

February 2012 Update on Feed Enforcement Activities to Limit the Spread of BSE

http://transmissiblespongiformenceph...e-on-feed.html



this is just part of it. but will give you a general idea. if anyone wants the long version, send me your email.......


kind regards,
terry
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  #27  
Old 03-30-2012, 07:19 PM
sandbur sandbur is offline
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thanks, Terry.

I'ts interesting that Spraker suggested they scrapie/CWD link from years ago at that one facility in Colorado.

Also thanks for the references concerning deer feed.

Minnesota's first 4 CWD locations were three elk farms and one deer farm. Two of the elk farms had a know link to each other.

I do not know if any tracebacks were shown to the Colorado elk facility.
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  #28  
Old 03-30-2012, 10:35 PM
Jeff23 Jeff23 is offline
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References regarding deer feed? Are you kidding me? Feed may have, possibly, maybe contained animal proteins that may have, possibly, maybe contained CWD or Mad Cow infected animals, that may have, possibly, maybe been fed to deer and elk. Good grief, a deer may have sniffed another deer's farts and inhaled the prion from the fecal molecules, too. Maybe a deer peed on my tire when I was out west once, and then my dog licked it off, then wiped his butt on my boots, which I then wore hunting, thereby infecting the entire state of PA. If only Scent Killer was also CWD killer, it all could have been prevented! Who was the author of those emails detailing all this possible ground breaking information? Oh..wait...I think I've seen that name before. Once again: good grief. You couldn't make this conversation up- truly stranger than fiction.
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Old 03-30-2012, 11:31 PM
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Quote:
Originally Posted by Jeff23 View Post
References regarding deer feed? Are you kidding me? Feed may have, possibly, maybe contained animal proteins that may have, possibly, maybe contained CWD or Mad Cow infected animals, that may have, possibly, maybe been fed to deer and elk. Good grief, a deer may have sniffed another deer's farts and inhaled the prion from the fecal molecules, too. Maybe a deer peed on my tire when I was out west once, and then my dog licked it off, then wiped his butt on my boots, which I then wore hunting, thereby infecting the entire state of PA. If only Scent Killer was also CWD killer, it all could have been prevented! Who was the author of those emails detailing all this possible ground breaking information? Oh..wait...I think I've seen that name before. Once again: good grief. You couldn't make this conversation up- truly stranger than fiction.




about that deer farting and another deer inhaling you claim.


http://www.plospathogens.org/article...l.ppat.1001257


i don't know how you deer farmers can stop that jeff23, i would just hate having to do a live rectal test period. maybe you inhaled too many of those farts



your stupid urine and cwd comments jeff23, well, that's a product of some of you deer farmers. so maybe you can tell us, how much of those deer and elk scents are being tested for CWD ???



That urine could serve as a natural route of infectious agent transmission. Exposure of surfaces to animal secretions and/or excretions can undoubtedly act as reservoirs for the infectious agent. Further, the detection of PASA in the urine of preclinical CWD deer indicates that the spread of the infectious agent can begin in non-symptomatic animals and therefore increases the risk for the continuous and unrealized spread of disease. Our studies may help in understanding the dynamics of horizontal spread of prion diseases in farmed and free-ranging animals.


http://jvi.asm.org/content/early/201....full.pdf+html




well jeff23, we know where you stand.

your either part of the problem, or part of the solution.

i think your colors have shown through brightly



kind regards,
terry
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  #30  
Old 03-31-2012, 06:24 AM
Jeff23 Jeff23 is offline
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Thanks, Terry, for your analysis, but if you're part of the solution- we need a new solution. If I, however, am part of the solution- then we are making progress compared to your decade-old scare tactics, selective-science-reporting, self-righteous methods. You have responded to so few of my direct questions to you, it has become funny-which led me to my recent silly posts. Did they seem funny to you? Odd? Welcome to the world of the rest of us reading your posts. Heck- you even went down a serious line with my last couple statements- you do understand sarcasm, right?
So much has changed in the last several years in the deer farming industry to improve every aspect of the business, many instances have been shown where other possible methods of transmission have been a bigger issue. I don't know as much about the scent industry, since that is very dissimilar to how we keep our deer. As the science expands, then that sector of the industry will more than keep up with the recommendations of those who are truly part of the solution- not some blogger who likes to tell everybody where to get off.
Now- here's where you interject a 40,000 character diatribe outlining how you emailed an official about how I've written such a pack of lies.
Oh- you still haven't outlined how the DNR would be better charged with handling captive cervids than the DofA, even after numerous people outlined the opposite based on reality. Just sayin'.
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